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Computational Biology

The aim of the Computational Biology team is to understand how individual bases in our genome predispose, alter and interact in normal and disease contexts.

Over the last decade, large international efforts have revolutionized our understanding of how our genome functions in normal and disease states.

The Computational Biology Team focusses on utilizing publicly available large datasets in combination with innovative computer algorithms to 1) improve diagnostic rates in children suffering from undiagnosed diseases and 2) discover new disease mechanisms and biomarkers in childhood cancers. 

Our aim is to understand how individual bases in our genome predispose, alter and interact in normal and disease contexts.

We focus on analyzing large public omics datasets using machine learning algorithms to understand dynamics of genome activity – which sequences are used when and where. Linking this data back to the genetic information of a patient is allowing us to discover the molecular causes of diseases and is key in predicting future disease and treatment outcomes.

Team Highlights - Rare diseases

  • Anderson, Denise, and Timo Lassmann. "An expanded phenotype centric benchmark of variant prioritisation tools." Human Mutation 43.5 (2022): 539-546.
  • Lassmann, Timo, et al. "A flexible computational pipeline for research analyses of unsolved clinical exome cases." NPJ genomic medicine 5.1 (2020): 1-11.
  • Anderson, D., Baynam, G., Blackwell, J. M., & Lassmann, T. (2019). "Personalised analytics for rare disease diagnostics". Nature communications, 10(1), 1-8.
  • Boycott KM, Hartley T, Biesecker LG, Gibbs RA, Innes AM, Riess O, Belmont J, Dunwoodie SL, Jojic N, Lassmann T, Mackay D. "A diagnosis for all rare genetic diseases: the horizon and the next frontiers". Cell. 2019 Mar 21;177(1):32-7.
  • Anderson, Denise, and Timo Lassmann. "A phenotype centric benchmark of variant prioritisation tools." NPJ genomic medicine 3.1 (2018): 1-9.

Team Highlights - Functional genomics

  • Hon, Chung-Chau, et al. "An atlas of human long non-coding RNAs with accurate 5′ ends." Nature 543.7644 (2017): 199.
  • Arner, Erik, et al. "Transcribed enhancers lead waves of coordinated transcription in transitioning mammalian cells." Science 347.6225 (2015): 1010-1014.
  • Forrest, Alistair RR, et al. "A promoter-level mammalian expression atlas." Nature 507.7493 (2014): 462.
  • Andersson, Robin, et al. "An atlas of active enhancers across human cell types and tissues." Nature 507.7493 (2014): 455.
  • ENCODE Project Consortium. "An integrated encyclopedia of DNA elements in the human genome." Nature 489.7414 (2012): 57.
  • Djebali, Sarah, et al. "Landscape of transcription in human cells." Nature 489.7414 (2012): 101.
  • Maida, Yoshiko, et al. "An RNA-dependent RNA polymerase formed by TERT and the RMRP RNA." Nature 461.7261 (2009): 230.

Featured research

Published in Science

Published in Nature

Team leader

Timo Lassmann
Timo Lassmann

BSc (Hons) MSc PhD

Feilman Fellow; Program Head, Precision Health and Head, Computational Biology

Team members (3)

Chin Wee

Chin Wee

PhD student

Timothy Chapman

Timothy Chapman

Research Assistant

Kathryn Farley

Kathryn Farley

PhD Candidate

Computational Biology projects

Featured projects

Centre for Advanced Cancer Genomics (CACG)

Current technologies to understand which genes are turned on or off only work on large amounts of biological samples. As a consequence all measurements we receive represent averages across multiple cell types present in the sample. The situation is comparable to studying the contents of a bowl of fr

Undiagnosed Diseases Program (UDP) and Bringing the benefits of precision medicine to children in Western Australia

We have started a project utilising whole genome sequencing of undiagnosed children living in WA to provide a definitive diagnosis. A major challenge here is that the role and functions of the inter-genic regions of our genome (the remaining 98%) are relatively poorly understood.

Computational Biology

Reports and Findings

Show all Computational Biology Reports and Findings

A corpus of GA4GH phenopackets: Case-level phenotyping for genomic diagnostics and discovery

The Global Alliance for Genomics and Health (GA4GH) Phenopacket Schema was released in 2022 and approved by ISO as a standard for sharing clinical and genomic information about an individual, including phenotypic descriptions, numerical measurements, genetic information, diagnoses, and treatments. A phenopacket can be used as an input file for software that supports phenotype-driven genomic diagnostics and for algorithms that facilitate patient classification and stratification for identifying new diseases and treatments.

Identifying SETBP1 haploinsufficiency molecular pathways to improve patient diagnosis using induced pluripotent stem cells and neural disease modelling

SETBP1 Haploinsufficiency Disorder (SETBD) is characterised by mild to moderate intellectual disability, speech and language impairment, mild motor developmental delay, behavioural issues, hypotonia, mild facial dysmorphisms, and vision impairment. Despite a clear link between SETBP1 mutations and neurodevelopmental disorders the precise role of SETBP1 in neural development remains elusive.

Use of privacy-preserving record linkage to examine the dispensing of pharmaceutical benefits scheme medicines to pregnant women in Western Australia

Medications are commonly used during pregnancy to manage pre-existing conditions and conditions that arise during pregnancy. However, not all medications are safe to use in pregnancy. This study utilized privacy-preserving record linkage (PPRL) to examine medications dispensed under the national Pharmaceutical Benefits Scheme (PBS) to pregnant women in Western Australia (WA) overall and by medication safety category. 

Time-course RNAseq data of murine AB1 mesothelioma and Renca renal cancer following immune checkpoint therapy

Time-critical transcriptional events in the immune microenvironment are important for response to immune checkpoint blockade (ICB), yet these events are difficult to characterise and remain incompletely understood. Here, we present whole tumor RNA sequencing data in the context of treatment with ICB in murine models of AB1 mesothelioma and Renca renal cell cancer. 

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