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Dr Linda Wijaya

Postdoctoral Researcher

Linda Wijaya

Postdoctoral Researcher

BSc PhD

linda.wijaya@thekids.org.au

+61 8 6319 1024

Dr. Linda Wijaya is a postdoctoral researcher at The Kids Research Institute Australia. She completed her PhD in Biomedical Science from Murdoch University in 2020. With over 18 years of experience in basic biology research and clinical trials, she has developed a broad range of expertise, mainly in biochemistry, cellular and molecular biology, and neuroimmunology.

Dr. Wijaya joined The Kids Cancer Centre in 2021. Her research interest is in neuroblastoma, a devastating cancer of immature nerve cells in children. Her work focuses on finding novel immunotherapy targets and developing new and safer treatment strategies for neuroblastoma.

Find Dr Wijaya on Google Scholar and ORCID.

Education and Qualifications

  • 2020: PhD in Biomedical Sciences – Murdoch University, Australia
  • 2006: BSc (Hons) in Developmental Biology – Bandung Institute of Technology, Indonesia

Awards/Honours

  • 2018: The IASP Pain Conference Financial Aid Awards
  • 2017: The Graduate Women WA Open Scholarship Award
  • 2016: Murdoch University Conference Travel Awards
  • 2014: Australian Postgraduate Award and Murdoch University Research Excellence Award
  • 2009: The Alzheimer’s Disease Conference Travel Fellowship

Active Collaborations

  • Prof Alistair Forrest – Harry Perkins Institute of Medical Research
  • Tumour Bank, Children's Cancer Research Unit, the Westmead Children’s Hospital
  • Nanostring Centre of Excellence in Spatial Biology, Griffith University Gold Coast Campus
Projects

Developing new immune based therapies for neuroblastoma

Neuroblastoma is a complex childhood cancer of the nerve cells and the most common solid tumour in children outside of the brain. The average age of diagnosis is 1-2 years and tragically 50% of children with high-risk neuroblastoma lose their battle within five years.

Published research

Inflammation induces α1-adrenoceptor expression in peripheral blood mononuclear cells of patients with complex regional pain syndrome

Persistent regional and systemic inflammation may promote pain and hyperalgesia in complex regional pain syndrome. In this study, we investigated whether stimulation of α1-adrenoceptors on peripheral blood mononuclear cells might contribute to this inflammatory state.

Stimulation of alpha-1 adrenoceptors may intensify cutaneous inflammation in complex regional pain syndrome

Alpha-1 adrenoceptors are overexpressed in the epidermis of a subgroup of patients with complex regional pain syndrome. Activating α 1 -adrenoceptors in epidermal cells increases production of the proinflammatory cytokine interleukin-6, a mediator of inflammation.